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GLP-1s are a class of drugs that enhance the effects of glucagon, which is a naturally occurring hormone in the body

Interruptions in therapy can negatively affect disease management, patient adherence, and continuity of care

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Excessive activation of autophagy will disrupt intracellular environmental homeostasis [92]

Microbiota-derived metabolites represent a plausible mechanistic interface: SCFAs and secondary bile acids can strengthen epithelial barrier function, modulate host metabolism via FXR- and TGR5-dependent signaling, and attenuate low-grade inflammation, potentially reinforcing pharmacologically targeted pathways